It wouldn't hurt people to do a little research on the description of
_clinical_ depression, its nature and its modern treatment. For example, it
is fairly well established that clinically depressed patients respond better
to a combination of psychotherapy (i.e. "understanding") _plus_
antidepressant drug therapy, than to either therapy alone. What you
fancifully refer to as 'chemical short circuiting', happens to be in
practice, 'what works'
Ken Collins wrote in message ...
>sometimes what appears to be 'apathy' is 'just' a rational outcome of
>done all that one can do in the face of 'difficulty'.
>>and =always=, the thing to do with what's been referred to as 'depression'
>is to acquire understanding... =never= to 'short-circuit' the nervous
>by attempting to chemically 're-engineer' it from outside.
>>there are other requirements which are outlined in AoK, Ap8.
>>Please Forgive me, Claude.
>>K. P. Collins (ken)
>>Claude de Contrecoeur wrote in message
><3796e8af.16349144 at nn-tk004.ocn.ad.jp>...
>>>>Int Clin Psychopharmacol 1997 Jul;12 Suppl 3:S29-33
>>>>Early onset of action of amineptine.
>>>>Green College, Oxford, UK.
>>>>A priority in the treatment of depression is to obtain rapid
>>an early stage. Since depressed patients, who are often convinced that
>>nothing can be done for them, may well have difficulty in adhering to
>>therapeutic management plan, they can be both uncooperative and
>>of treatment measures. The rapid correction of this often resigned
>>is an ESSENTIAL aspect of treatment. According to a variety of
>>criteria, amineptine often achieves RAPID improvement, particularly on
>>measures of psychomotor retardation. Initially, antidepressant
>>is an essential measure in the relief of depressive symptoms, although
>>subsequently, it may also become a complement to psychotherapeutic
>>Amineptine has been shown to act directly on the dopaminergic pathway,
>>unlike other antidepressants, which act on this system only via their
>>effects on the serotonergic or noradrenergic systems.