I recently received a mail reply from Kay Klier concerning the activity
of P.I. on skin of different thickness or callosity. It was a very logical
reply. I have a question. Does anybody know why that would be the case?
Is it not strictly a surface phenomenon? Does the toxin have to penetrate
the outer layer because it consists of "dead cells"? How does it penetrate
the outer layer? Through cracks, pores, what?
vsummers at polaris.cv.nrao.edu