which varies in magnitude, for similar noise exposures, between different
Where a large loss has been sustained by a previously normal-hearing
individual, following substantial but not extreme noise exposure, are we
usually content to explain this as undue sensitivity to noise - the extreme
end of a "normal" distribution so to speak - or does anyone have examples
of this occurring due to some pathological process (in addition to noise) -
some form of infection maybe? Any ideas?