Telomerase seems to be the hot topic around here. It would certainly be an
easy solution to the aging problem, if only it were that simple. I think
the evidence supporting the role of telomere shortening leading to in vitro
senescence is overwhelming and people take this as a given now. Most
academic researchers (see Martin J. of Gerontology: Biological sciences,
vol 48, No 5, B171-172) however, that is researchers who do not depend on
gulible investors for funding, do not share the believe that in vitro
senescence, hence telomere shortening, has anything to do with organismal
aging.
I reviewed the evidence 2 years ago, and I did not find any compeling
argument supporting the statement that in vitro senescence has any
relationship to in vivo aging.
Perhaps data has come out to support this point within the last 2 years. In
this case, will anybody please enligthen me!
