That seemed very thorough and certainly interesting. I'm not sure I'd
be quite so dismissive, though. Considering ageing as a multitude of
entropic mechanisms of different relative importances acting upon the
organismal genome, it is not necessary that the subtle phenotypic
differences in ageing be correlated to differences (or similarities) in
molecular processes. It is possible that longer-lived mammals make use
of telomerase repressor lifespan regulation whereas some other primary
mechanism is more evolutionarily appropriate for, say, murine species.
